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Placebo and control groups demonstrated significantly increased immobility time in the forced swim test, which was reversed with IN-BDNF
2), aiming to enhance and/or complement the effect of GLP-1 agonism on weight and metabolism [14]
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preventing transcriptional activity of nuclear factor kappa-light-chain-enhancer of activated B cells (NF-B) that regulates downstream transcription of a vast range of cytokines
