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Together these findings suggest that ALCAR is a potent neuroprotectant, facilitated by its ability to cross the blood brain barrier
The partial rescue of the above-described symptomatology can be ascribed to the activity of other transporters such as ATB 0,+ (SLC6A14), MCT9, and probably, OCTN1 (SLC22A4) that accept carnitine with a much lower affinity if compared to OCTN2
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