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Ginsenosides Rg1, Rg3, and panaxadiol saponins have been found to inhibit the ROS-induced release of matrix metalloproteinases and degradation of the fibrous wall of atherosclerotic plaques and the basement membrane of endothelial cells, thus weakening the physical destruction caused by atherosclerotic plaques (Bao et al., 2009

Ethanol treatment led to marked lipid dysregulation in alveolar macrophages, which significantly impaired the glycolytic response to LPS, thus weakening the cells ability to mount effective immune responses