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Upon entering the cell, intracellular proteases cleave this complex, leading to liberation of Cbl

Numerous studies have demonstrated that HER2 inhibition can induce mitochondrial dysfunction and disrupt adequate energy supply (Figure 1) ( 2.7 Other possible mechanisms of cardiotoxicity The study conducted by Huan Wang et al

This is indicated by increased serum creatinine and an increased risk for diabetes in G6PD-deficient subjects (28, 74, 90, 108, 109) and albuminuria and metabolic dysfunction in deficient animals (40, 111, 115), which could have adverse cardiac effects