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470 In animal models of Apelin-13-caused cardiomyocyte hypertrophy and heart failure, NCOA4-activated ferritinophagy leads to the upregulation of SFXN1 expression, causing mitochondrial iron overload and mitochondrial ROS overproduction, increased lipid peroxidation, and ultimately, ferroptosis
When conversion is impaired, patients may experience low tissue-level T3 despite normal serum TSH and T4 values
Therefore, analysis of NRF2 target genes based on patient stratification with these mutations allows us to identify novel genes, biomarkers, and therapeutic targets in lung cancer
