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doi: 10.1093/hmg/ddh094

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While hypoperfusion likely plays a role, oxidative stress and subsequent renal tissue damage from reactive oxygen species occurs due to both ischemia-reperfusion injury and activation of inflammatory cascades when blood is exposed to the CPB circuit [37]

Molecular insights into nitrogenase FeMoco insertion: TRP-444 of MoFe protein -subunit locks FeMoco in its binding site