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[DOI] [PubMed] [Google Scholar] 39.Tanase D.M., Gosav E.M., Costea C.F., Ciocoiu M., Lacatusu C.M., Maranduca M.A., Ouatu A., Floria M
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The delay in clearance of apoptotic cells may prolong interaction between ROS and nuclear debris and generate neo-epitopes that subsequently stimulate broad spectrum of autoantibody formation leading to inflammation and organ damage in SLE [5]
