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This makes it biologically plausible that changes in NO availability could affect both migration and angiogenesis-associated endpoints

Likely, with regular eating/drinking, this indicates an always alerted stomach that should be further basis also for the classic Roberts stomach cytoprotection/adaptive cytoprotection concept (1-3) functioning

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While human trials are limited, mechanistic research suggests several pathways: Promotes new blood-vessel formation (angiogenesis) and improved micro-circulation in injured tissue