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Of note, the treatment of BERK SCD mice microglial cells with TLR4 inhibitors reversed this effect [125]
Deficient glutamate clearance, usually associated with downregulation of EAAT transporters is one of the primary mechanisms of glutamate excitotoxicity leading to neuronal death (see Table 2 and the previous section)
It works through a different upstream receptor system
9c), without causing a depletion in glutamate (Extended Data Fig
