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Key Research Effects Upregulates genes involved in collagen I & III synthesis Activates fibroblasts, keratinocytes, and endothelial cells Enhances angiogenesis and wound closure Suppresses NF-Bmediated inflammation Reduces oxidative stress via SOD activation Transcriptomic studies show GHK-Cu can positively regulate thousands of genes associated with tissue repair while downregulating pro-fibrotic and inflammatory pathways

10.1172/JCI67230 68 H

Regular monitoring may be appropriate, especially if symptoms of deficiency develop

Involvement of endoplasmic reticulum Ca2 + release through ryanodine and inositol 1,4,5-triphosphate receptors in the neurotoxic effects induced by the amyloid-beta peptide