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Note: The above statements have not been evaluated by the FDA (U.S
The accumulation of A, as well as oxidative stress and other mechanisms of excitotoxicity, can lead to canonical activation of calpain through significant increases of Ca 2 + in the cytoplasm after NMDAR activation [59]
Callaci, K.L
The genes exhibiting differential expression in the lipid and atherosclerosis pathways include APOB ( p = 0.008), CD36 ( p = 0.040), CALM1, CALM2, CALM3 ( p = 0.015), and HSPA8 ( p = 0.047)
