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86 Recent data shows that excessive free radicals in the brain enhance oncogenesis by encouraging initiation, proliferation, angiogenesis, invasion, and cell arrest, regulating numerous processes that support oncogenesis

First, KEAP1 has been shown to have complex interactions with various other proteins besides NRF2 (Hushpulian et al., 2021) and concerns about potential side effects arise from the diverse array of cellular processes covered by KEAP1 interactor proteins, involving DNA replication, cytoskeletal dynamics, transcription, and apoptosis