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Description
Glutathione depletion and a corresponding increase in reactive oxygen species (ROS) during microbial infection is a key driver of the immune response and inflammation
This release of FeSOD-A into the cytosol or the inhibition of FeSOD-A import into the mitochondria makes the mitochondria even more susceptible to oxidative stress due to the accumulation of ROS
Induction of ferroptosis in MM cells upregulates key genes implicated in cellular stress, death, inflammation, and fatty acid metabolism, including ferritin heavy chain 1 (FTH1), HO-1, and SLC7A11
He reviewed her U/S pictures with me explaining the anatomy
