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In double knockout mice lacking the NOX1, NOX2, or NOX4 genes alongside the BH 4 gene, respectively, the bioavailability of NO and BH 4 in endothelial cells is increased, DHFR content is significantly increased, and the incidence of abdominal aortic aneurysms is significantly decreased, as compared with mice with knockout of the BH 4 gene only
Myocardial PD-L1 expression in patients with ischemic and non-ischemic heart failure
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