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This is a research schedule, not a prescription

In particular, the animal study 7 in which pharmacological inhibition of GPx activity significantly reduced brain infarct damage suggested that under reduced oxygen supply, H 2 O 2 may exert either physiological or protective roles in the brain through its metabolic degradation to oxygen by catalase compensating for the lack of oxygen that occurs in the tissue after an ischemic event

While promising, a multi-target strategy must also consider the potential risks associated with simultaneous targeting, particularly regarding its effects on normal tissue homeostasis

San Diego, California, January 25, 2009