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Chromatin immunoprecipitation (ChIP) demonstrated increased occupancy of H2AX and decreased occupancy of all three acetylation marks at DSBs in both IDH1 inhibitor-treated and TMLHE-knockdown cells (Fig
Abnormal mitochondrial morphology can inhibit mitochondrial OXPHOS and alter calcium homeostasis and mitochondria-mediated apoptosis, leading to mitochondrial dysfunction and mitochondria-related diseases (91)
