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Description
There's no real debate here
93rd Annual Meeting of the German Society of Oto-Rhino-Laryngology Head & Neck Society, DE
However, under oxidative stress condition, specific cysteinyl residues of KEAP1 are modified, thereby preventing the binding of KEAP1 to NRF2
These cells, which accumulate with age or stress, contribute to organ dysfunction by promoting chronic inflammation and impairing tissue repair
