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PMID 19699272

BPC-157 has been investigated for interactions with VEGFR2-, ERK1/2-, eNOS-, and FAK-associated signaling networks together with regulatory processes involving extracellular protein systems

The aim of this selective compilation of a large body of research is to highlight the chemically plausible mechanisms through which some metals that are unable to react by the radical pathways related to the Fenton or Haber-Weiss reaction, are nevertheless able to generate a physiological damage by triggering oxidative stress through their reactivity towards the soluble thiolome pool
