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Antibiotics including rifaximin and metronidazole have been investigated for their capacity to inhibit TMAO formation by targeting gut bacteria that create TMA
Administration of MS-275, an HDAC inhibitor, also increases expression of mGlu2 receptors, supporting the role of epigenetic mechanisms in the regulation of mGlu2 receptors [30]
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Recent advances in the modulation of pain by the metabotropic glutamate receptors
