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Retinal circadian clocks are major players in the modulation of retinal functions and photoreceptor viability

PMID 11035031

The first evidence that eNOS uncoupling may be a pathophysiologically relevant phenomenon was provided by in vitro studies in which it was demonstrated that native low-density lipoprotein 81 and even more pronounced oxidized low-density lipoprotein 82 are able to stimulate endothelial superoxide production and that this phenomenon is inhibited by the NOS inhibitor N G -nitro-L-arginine methyl ester, pointing to a causal role for eNOS in superoxide production

Then, we found mtDNA transfection accelerated the levels of ferroptosis biomarkers and reversed the inhibitory effects of SP on the ferroptosis process in TNF--induced Caco-2 cells