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Exosomes derived from LPS-preconditioned bone marrow-derived MSC modulate macrophage plasticity to promote allograft survival via the NF-B/NLRP3 signaling pathway

In addition, MAO-generated ROS also lead to p53 activation and consequent mitochondrial oxidative damage due to the inhibition of parkin-mediated mitophagy by p53 [31]

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