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In addition, hyperoside exhibits a novel therapeutic potential for diabetic nephropathy by attenuating AGE/RAGE binding and TGF-1 expression through the inhibition of pERK1/2, p38 MAPK, and IB phosphorylation (Zhang et al., 2013

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This knowledge gap stems not from the complexity of biochemical reactions and the metabolic crosstalk in dynamic biological settings but from technical limitations in investigating metabolism at single-cell resolution

Antioxidants have been explored to an extent with regard to their ability to counteract the effects of lipid peroxidation as a result of chemotherapy, which makes it possible to minimise the side effects of the treatment while keeping the tumour -fighting properties of the drugs unchanged [11]