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Severe asthma in children is characterized by altered airway and intracellular AM GSH homeostasis that translates to impaired AM function

Although WT -GCS-GS is nearly uninhibited by GSH (Ki 140 mM), shorter -GCS domain constructs were unexpectedly found to be strongly inhibited (Ki 15 mM), reproducing a physiologically important regulation seen in monofunctional -GCS enzymes