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Description
[(accessed on 25 November 2020)]
This presents as abdominal pain, distension, nausea, vomiting, constipation, and high-pitched or absent bowel sounds
Targeting other KRAS mutations and indirect strategies Targeting other KRAS mutations The successful development of KRAS (G12C) inhibitors is dependent on the covalent inhibition of cysteine and high GTPase activity of KRAS (G12C), which is not fully present in other KRAS-mutant types 114
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