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However, the survival benefit of sorafenib was disappointing in clinical trials, owing to its low response rate and short effective duration, caused by intrinsic and acquired resistance (Llovet et al., 2015)

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However, for the benchmark datasets, which includes only predictions within 2 pKa units of each other, the opposite was noticed, namely that the basic dataset showed better concordance with the OPERA, XGB and DNN models

As a result, the increased secondary BAs can activate TGR5, which increases L-cells and GLP-1 secretion by inhibiting the RhoA/ROCK pathway and inducing YAP activation [157, 203]