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Protective effects of hesperidin and diosmin against acrylamide-induced liver, kidney, and brain oxidative damage in rats

Overcoming barriers in cancer biology research: current limitations and solutions

11 Although botulinum toxin's potent effects on ACh release inhibition suggest it is the signaling neuropeptide behind the flushing, another peptide must also be involved because atropine blockade of ACh action in patients with Frey syndrome stops the sweating, but not the flushing

Therefore, the seemingly additive effects of CgCS and CitS may not be simply explained by their cumulative positive effects on glutamate production