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Recently Ma et al., using multiparametric analysis of GPX4 and Apoptosis Inducing Factor Mitochondria Associated 2 (AIFM2) also called ferroptosis suppressor protein (FSP1) expression and function identified high expression of both proteins as novel biomarkers of pejorative prognosis in AML [35]

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This study also highlighted a promise of NLCs in the treatment application of AD via an intranasal route, as evidenced by increased accumulation and prolonged retention time of quercetin-loaded NLCs in the nasal cavity (~8 h) (Satpathy et al., 2025)

Their primary mechanism of action consists in the inhibition of the COX enzyme, which is essential for the synthesis of prostaglandins (PGs) [68]