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For example, the ingestion of trimethylamine-containing (TMA-containing) dietary nutrients enriched in animal products such as choline, phosphatidylcholine, and l-carnitine can serve as precursor for the ultimate generation of an atherogenic metabolite, trimethylamine N -oxide (TMAO), via a metaorganismal pathway involving initial gut microbial formation of TMA, followed by host hepatic conversion of TMA into TMAO (48)

[DOI] [PMC free article] [PubMed] [Google Scholar] 146.Weiskopf K, Jahchan NS, Schnorr PJ, Cristea S, Ring AM, Maute RL

Under most physiological conditions, endogenous carnitine biosynthesis may be sufficient to maintain adequate tissue concentrations ( Reference Ozorio 30 ) , and therefore dietary supplementation with d-carnitine might only be partially able to affect l-carnitine concentration in particular tissues

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