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The direct contact needed for this interaction support the pathogenetic role of increased intestinal permeability in contributing to VOC in SCD
GHS-R deletion decreases dopaminergic activity in the ventral tegmental area (VTA) by down-regulating DAT, but increases dopaminergic activity by up-regulating the expression of both dopamine synthesis enzyme (TH) and DAT in SN of HFD-fed neuronal-specific GHS-R knockout mice 9
