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Description
doi: 10.4239/wjd.v12.i6.794 41 Corredor-RengifoD.Tello-CajiaoM
The primary mechanisms proposed for this phenomenon include oxidative damage to DNA, lipids, and proteins that initiates the carcinogenic process and the exacerbation of oxidative stress that disrupts antioxidant defense systems and associated signaling pathways, thereby facilitating abnormal cellular proliferation
Bogdanova OV, Kanekar S, DAnci KE, Renshaw PF
It is not intended for human use, clinical diagnostics, or any form of therapeutic application
