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Description
Mechanisms of mitochondrial ROS overproduction in NAFLD: Mitochondria are a central source of reactive oxygen species (ROS) in non-alcoholic fatty liver disease (NAFLD), primarily due to electron leakage at complexes I and III of the electron transport chain (ETC)
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At these pH values, the peptide molecules carry minimal net charge, which reduces the electrostatic repulsion that normally keeps individual molecules separated
This process is mediated by G-protein-signaling, activating phospholipase C and increasing cytosolic Ca 2+ concentrations
