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Enhanced glycolysis-derived lactate promotes microglial activation in Parkinsons disease via histone lactylation

Key Research Effects Upregulates genes involved in collagen I & III synthesis Activates fibroblasts, keratinocytes, and endothelial cells Enhances angiogenesis and wound closure Suppresses NF-Bmediated inflammation Reduces oxidative stress via SOD activation Transcriptomic studies show GHK-Cu can positively regulate thousands of genes associated with tissue repair while downregulating pro-fibrotic and inflammatory pathways

Delivery of adipose-derived precursor cells for peripheral nerve repair

Citrulline depletion by ASS1 is required for proinflammatory macrophage activation and immune responses