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Frameshift mutants of amyloid precursor protein and ubiquitin-B in Alzheimers and Down patients
However, the average neutrophil count in the control group was significantly lower than in both the zinc oxide and TCC groups at all time points
A dysregulation of brain cholesterol homeostasis may be involved in the pathogenesis of Alzheimers disease [2] through interference with the amyloidogenic A pathway [3], impairment of cerebral blood flow [4], and other mechanisms [5]
