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This action, combined with the induction of GLT1 in astrocytes, may at least in part account for the beneficial effects of NAC in conditions associated with excessive release of glutamate and impairment of glutamate homeostasis, and characterized by maladaptive synaptic plasticity and excitotoxic neuronal death [113]

System Xc , GSH synthesis, and GPX4 have been shown to protect against cell death triggered by multiple oxidative stress responses

If this subtle approach does not resolve the problem, other possible approaches include changes in the pharmaceutical form for oral administration, weekly oral ingestion of LT4 under the supervision of medical staff, or exceptionally (and preferably at an expert centre) the parenteral infusion of LT4