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Therefore, lots of combinatorial strategies have been established to provide improved clinical results with the aim of creating a hot TME by increasing PD-L1 expression, enhancing TIL infiltration, and targeting other type of cells in the TME

Despite their reduction through macrophage phagocytosis, the inflammatory response persists due to the continued macrophage engagement with MSU crystals (52), contributing to the chronic inflammation characteristic of HUA

GN binding to phospholipids leads to phospholipidosis, oxidative stress, inflammation, and necrosis, compromising membrane integrity 8,10

To assess depression-like phenotypes, we employed the sucrose preference test