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Persistent DNA damage can promote apoptosis of follicular cells, leading to the release of self-antigens such as TPO and thyroglobulin within the cells, thereby disrupting immune tolerance (Fig

BPC-157 acts primarily at the tissue and gut level - more localised, faster-acting on specific injury sites and gut lining

Structure and function of the xenobiotic substrate binding site of a glutathione S-transferase as revealed by X-ray crystallographic analysis of product complexes with the diastereomers of 9-(S-glutathionyl)-10-hydroxy-9,10-dihydrophenanthrene

A family with erythrocytosis establishes a role for prolyl hydroxylase domain protein 2 in oxygen homeostasis