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Even so, the effect does not always persist long term and the magnitude can be modest
Taken together, the studies on antioxidant enzymes in AD demonstrated no consistent major deficiencies in their detoxifying processes, consistent with the idea that the mechanism of neuron death in AD is likely not a failure of these defenses against free radicals
Peroxisome proliferator-associated receptor (PPAR) pathways, particularly PPAR-beta, are implicated in controlling these inflammatory processes
