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By inhibiting the folate cycle and de novo purine synthesis, MOTS-c triggers an AMPK-driven shift that encourages the body to burn fuel rather than store it
Mechanistically, stabilization of wild-type p53 and induction of the p53 target gene CDKN1A (p21) leads to decreased expression of the ribonucleotide reductase (RNR) subunits RRM1 and RRM2
