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Similarly, in T cell-driven graft-versus-host disease (GVHD), FXR agonism exacerbated mortality, while T cell-specific FXR deletion or UDCA administration (again exhibiting FXR antagonistic properties) improved outcomes (88)

If a significant deficiency is left untreated over time, some of this nerve damage can become permanent, which is why prompt evaluation matters

GLUT4 expression is also found in the brain and the heart

From this research it seems that the best explanations may be through the potential reset of a disrupted methionine-folate cycle and aiding in the removal of toxic metals and chemicals from the brain