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Description
The evidence that OTA caused increased oxidative stress led to the expectation that gene expression profiles after exposure to OTA in animals would show the induction of many genes responsive to exposure to oxidative stress
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There is also formal clinical investigation, including the cystic fibrosis trial noted earlier
Plant Cell 15, 939951
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PKM2-dependent metabolic skewing of hepatic Th17 cells regulates pathogenesis of non-alcoholic fatty liver disease
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