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doi: 10.1038/s41531-023-00505-0 65 HerbertM
While the direct evidence for MOTS-c longevity extension is still emerging, its mechanisms are highly promising
Reduced ATP levels impair the function of ionmotive ATPases such as Na/KATPase, resulting in membrane depolarization, overactivation of voltagegated Ca channels, and ultimately cytotoxic Ca overload [110]
Another strategy relies on the redistribution of AQP4, which is at least in part regulated by calmodulin
