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Electrophysiological studies in cortical and hippocampal neurons have shown that exogenous glycine or D-serine can further potentiate NMDA receptormediated currents, indicating that endogenous co-agonist concentrations remain below saturating levels at least in a subset of synapses (Wilcox et al

Tanshinone IIA can inhibit focal death by modulating Txnip/NLRP3 inflammatory vesicles, regulation of TGFB1 inhibits high glucose-induced inflammation and cellular pyroptosis in renal tubular epithelial cells, may promote defense against DKD by inhibiting apoptosis caused by intracellular oxidative stress, can prevent podocyte damage in DKD by promoting autophagy and inhibiting inflammation, in part by inhibiting the PI3K/Akt/mTOR signaling pathway (Li et al., 2024), also ameliorates DKD-induced renal fibrosis by regulating miRNA-34a-5p in in vitro and in vivo studies