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All pathological processes, except for Canavan disease, lead to decreases of its concentration
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Investigations assess its potential effects on cuprizone-associated demyelination, traumatic brain injury, neurotoxin-induced dopaminergic disruption, neurotransmitter regulation, and peripheral or central nerve regeneration
Mitochondrial dysfunction, a key feature of AD, further exacerbates ROS production, impairs ATP generation, disrupts calcium homeostasis, and ultimately induces neuronal apoptosis (Bhatt et al., 2021)
