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glutathione oxalates Oxalate (dys)Metabolism: Person-to-Person Variability, Kidney and Cardiometabolic Toxicity Frontiers | The significance of
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doi: 10.1093/carcin/1.10.813

JNK activation and translocation to mitochondria mediates mitochondrial dysfunction and cell death induced by VDAC opening and sorafenib in hepatocarcinoma cells

It was developed alongside Semax at the Russian Academy of Sciences and has been approved in Russia as an anxiolytic medication

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