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Exposure to METH during pregnancy can disrupt the offspring's gut microbiota, serum metabolome, and BA homeostasis, and maternal inulin supplementation can reverse the disruption of the gut microbiota and serum metabolome in the offspring induced by METH (Li et al

If the topic under investigation is angiogenesis or localized cell signalling then it could very well lead to using BPC-157, but if the research revolves around cell migration and actin then TB-500 might seem more appropriate due to its association with a completely different pathway

Restoring mitochondrial biogenesis with metformin attenuates -GP-induced phenotypic transformation of VSMCs into an osteogenic phenotype via inhibition of PDK4/oxidative stress-mediated apoptosis