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Global hypoxia affects the electron transport chain, fatty acid beta-oxidation and other metabolic pathways essential for mitochondrial energy production

Malonyl-CoA generated by acetyl-CoA carboxylase - isoform 2 (ACC2) binds with high affinity to each of the CPTI isoforms and acts like a switch to control the sensitivity to this key inhibitory molecule and regulate the transfer of fatty acids into the mitochondrial matrix and thence their oxidation

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Two ubiquitin ligases, MAFbx and MuRF1, have previously been shown to be rapidly induced in multiple models of muscle atrophy, including the TNF--induced muscle wasting model [14, 32]