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Description
Such injuries, characteristic of various neurological disorders like cerebral hemorrhage, are predominantly driven by oxidative stress, inflammatory cascades, and cellular death [161]
We therefore propose that for a better understanding of ME/CFS, micro- and macro-circulatory failure, endothelial dysfunction, metabolic dysfunction, redox imbalance, inflammatory stimulation, and CNS inflammation need to be jointly considered (see Figure 2)
The contribution of inducible nitric oxide and cytomegalovirus to the stability of complex carotid plaque
Such features were also reported in C
