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INTRODUCTION As one of the driving forces behind the cellular detoxification machinery, glutathione S -transferases (GSTs) and especially the pi class glutathione S -transferase P1 (GSTP1) is currently in the focus of the cancer research community, evaluating the relevance of GSTP1 epimutations for cancer development and its potential as a major epigenetic cancer biomarker

J.KollmannsbergerC.MetznerB.HartmannJ

GSH prevented the accumulation of platinum in the DRG as well as chelating aluminum from the brain and serum [44,45]

Taken together, these findings support a model in which mitochondrial and cytosolic Src act as non-canonical mediators of cardioprotection, distinct from RISK or SAFE pathways, but which can interact, at least with RISK/NO pathways [55]