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Therefore, evidence for metabolic failure during disease progression (after diagnosis) comes from functional studies conducted in ALS patients, postmortem studies of ALS patient tissue, and mouse models
Schobersberger, W., Hoffmann, G
Insulin binds with high affinity to INSR-A and INSR-B but also binds with a lower affinity to IGF1R and the hybrid receptors INSR-A/IGF1R and INSR-B/IGF1R [14, 15]
tuberculosis (Zheng et al., 2017)
