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These findings suggest that the role of type II membrane-bound CD38 is to maintain NAD + homeostasis by regulating precursors of NAD + synthesis in the extracellular environment, thus calling into question the veracity of a topological paradox

In this review, we focus on the genetic, molecular, and cellular mechanisms driven by HGF-MET in the nervous system, starting from their involvement in brain development and ending to their implication in neurodevelopmental disorders (NDDs)

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